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Fatigue is a symptom of mild deficiency of zinc so perhaps Fibromyalgia and Chronic Fatigue could be helped with the intake of foods containing zinc. Fatigue is a widespread
problem in the world even without chronic illness. Zinc is one of those minerals our bodies do not produce and we need to be replenished by our food intake or supplement. Zinc does so
much yet we hear so little about it. Perhaps because it got a bad wrap from the FDA but if you read the whole story “The Stink about Zinc” you will see that it was based on the topical
use of zinc in a nasal spray that they think caused a loss of smell. Zinc is vital to your well being.
http://www.jonbarron.org/immunity/bl1007029/health-blog-zinc-boost-immune-system
In the book The Healing Foods they link zinc and acne, colds and wound healing.
Zinc: The Healing Mineral
- part of certain enzymes some of which metabolize protein, carbohydrate and alcohol
- helps in making protein and building bones
- affects the senses of smell and tastes
- aids in healing wounds
Symptoms of mild deficiency are:
- poor appetite
- supoptimal growth
- reduced sense of taste and smell
- scaly skin
- delayed wound healing
- fatigue
- hair loss
- diarrhea
- reduced resistance to infection
Some have special need to zinc.
- acrodermatitis enteropathica
A number of chronic health problems may interfere with zinc nutrition, these include:
- alcoholism
- chronic infections
- inflammatory diseases
- diabetes
- kidney disease
- pancreatic disease
- psoriasis
- certain kinds of anemia
Large amounts of zinc may be lost due to
- surgery
- burns
- multiple injuries
What do we eat?
Good
1 cup cooked beans
2 slices rye bread
2 slices whole wheat bread
1/2 chicken breast
4 oz chicken meat
4 to 5 hard-shell clams
8 oz surf clams
1 cup cottage cheese
1 oz 40 Bran cereal
1 cup lentils
1 cup lima beans
1 cup lobster meat
1 cup milk
1 cup oatmeal
1 oz puffed oat cerial
1 cup peas
1 medium baked potato
1 cup brown rice
4 oz salmon
7 oz spaghetti and meatballs
1 cup spinach
1 oz shredded wheat
4 oz white turkey meat
1 tablspoon toasted wheat germ
4 oz whitefish fillet
1 cup plain low-fat yogurt
Better
4 oz lamb
4 oz lean pork
4 oz dark turkey meat
Best
4 oz lean beef
1 cup crabmeat
1 cup cowpeas
4 oz oysters
Cowpeas?? What the heck are cowpeas? : )
Food Chem Toxicol. 2002 Aug;40(8):1113-7.
CSIRO Health Sciences and Nutrition, PO Box 10041, Gouger Street, BC, SA, 5000, Adelaide, Australia. michael.fenech@hsn.csiro.au
Diet as a key factor in determining genomic stability is more important than previously imagined because we now know that it impacts on all relevant pathways, namely exposure to dietary carcinogens, activation/detoxification of carcinogens, DNA repair, DNA synthesis and apoptosis. Current recommended dietary allowances for vitamins and minerals are based largely on the prevention of diseases of deficiency such as scurvy in the case of vitamin C. Because diseases of development, degenerative disease and aging itself are partly caused by damage to DNA it seems logical that we should focus better our attention on defining optimal requirements of key minerals and vitamins for preventing damage to both nuclear and mitochondrial DNA. To date, our knowledge on optimal micronutrient levels for genomic stability is scanty and disorganised. However, there is already sufficient evidence to suggest that marginal deficiencies in folate, vitamin B12, niacin and zinc impact significantly on spontaneous chromosome damage rate. The recent data for folate and vitamin B12 in humans with respect to micronucleus formation in blood and epithelial cells provide compelling evidence of the important role of these micronutrients in maintenance of genome integrity and the need to revise current RDAs for these micronutrients based on minimisation of DNA damage. Appropriately designed in vitro studies and in vivo placebo controlled trials with dose responses using a complementary array of DNA damage biomarkers are required to define recommended dietary allowances for genomic stability. Furthermore these studies would have to be targeted to individuals with common genetic polymorphisms that alter the bioavailability of specific micronutrients and the affinity of specific key enzymes involved in DNA metabolism for their micronutrient co-factor. That there is a need for an international collaborative effort to establish RDAs for genomic stability is self-evident.
PMID: 12067572 [PubMed - indexed for MEDLINE]
Article from: http://www.ncbi.nlm.nih.gov/pubmed/12067572